Blaming Overeating On Drinking

You know what makes you fat? It’s not food. It’s drinking alcohol. Wait, what? Yup, according to a study financed by the NIH, conducted by the Indiana Alcohol Research Center, and published earlier this year in the journal Obesity, researchers claim that what they’ve dubbed “the apéritif phenomenon” may be causing our obesity epidemic. Except that they’re not.

The self-described “internationally recognized news website” Inquisitr, under the category “Celebrity Health,” published an article entitled “Alcohol Sensitizes Brain’s Response To Food Aromas, Say Scientists — Is Liquor Responsible For Rising Obesity?” Naturally, Alcohol Justice gleefully tweeted the bad news as “new evidence points to alcohol’s role in U.S. obesity epidemic.” Except that, as I mentioned, the evidence does nothing of the kind.

The study that the article is based on is entitled The apéritif effect: Alcohol’s effects on the brain’s response to food aromas in women. Here’s the abstract:

Consuming alcohol prior to a meal (an apéritif) increases food consumption. This greater food consumption may result from increased activity in brain regions that mediate reward and regulate feeding behavior. Using functional magnetic resonance imaging, we evaluated the blood oxygenation level dependent (BOLD) response to the food aromas of either roast beef or Italian meat sauce following pharmacokinetically controlled intravenous infusion of alcohol.

BOLD activation to food aromas in non-obese women (n = 35) was evaluated once during intravenous infusion of 6% v/v EtOH, clamped at a steady-state breath alcohol concentration of 50 mg%, and once during infusion of saline using matching pump rates. Ad libitum intake of roast beef with noodles or Italian meat sauce with pasta following imaging was recorded.

BOLD activation to food relative to non-food odors in the hypothalamic area was increased during alcohol pre-load when compared to saline. Food consumption was significantly greater, and levels of ghrelin were reduced, following alcohol.

An alcohol pre-load increased food consumption and potentiated differences between food and non-food BOLD responses in the region of the hypothalamus. The hypothalamus may mediate the interplay of alcohol and responses to food cues, thus playing a role in the apéritif phenomenon.

The Indiana Alcohol Research Center “focuses on the elucidation of the biomedical and psychosocial factors that contribute to alcohol abuse and alcoholism,” which suggests to me they’re another group like the NIAAA, or National Institute on Alcohol Abuse and Alcoholism (whose grant created the IARC), is exclusively interested in exploring the negative aspects of alcohol. And just like the NIAAA, it’s right there in their charter.

Curiously, yesterday the full text of the article was also online, but today it’s restricted. They start with the premise that “consuming alcohol prior to a meal (their “apéritif phenomenon”) increases food consumption,” but of course that’s the point of an apéritif, or at least to enhance and make the experience of the food and/or the food and the drink better.

But as they conclude, this “pre-loading” of alcohol is what makes us want to eat more, which they believe that their study shows. When I briefly looked at the entire article, their longer discussion of the findings, as is quite common, suggested caution in drawing too many conclusions and suggesting further study was warranted. As the shorter conclusion states, these “food cues” play “a role in the apéritif phenomenon,” which is not exactly the same as saying “drinking is responsible for American obesity.”

But that didn’t stop author Alap Naik Desai from making such speculation, fueling the prohibitionist response that of course “Liquor [is] Responsible For [the] Rising Obesity” in the United States.

A research conducted by Indiana University indicated that exposure to alcohol enhanced the brain’s sensitivity and heightened its response to food aromas. In simpler words, food seemed much more appealing and appetizing, which, of course, led to extra consumption. Connecting the dots, one could also summarize that alcohol consumption was responsible for increased intake of food and hence a hidden cause of obesity.

I’m not sure which dots he’s referring to, since that’s a fairly absurd statement that isn’t contained in the study itself. But beyond that, the study involved just 35 female test subjects, no men at all. And it seems hard to extrapolate anything meaningful that could be applicable to the human population from so few people. Also, they claim that people “responded enthusiastically to food aromas after the body had been exposed to alcohol,” but not from drinking it, simply from having smelled it. Despite the lack of causation, or a robust sample size or even anything resembling reality, the lead author of the study, William Eiler, apparently told Desai that “this poses a major risk to those trying to keep their weight down.” Seriously, “a major risk” because 35 women seemed more hungry after sniffing alcohol? Desai continues. “With America weighing down under an obesity epidemic and two out of every three American adults consuming alcohol, there is an immediate need to find more connecting factors between the brain, food, and alcohol, advise the scientists.”

Except that this idea is easily demolished by one simple fact. Even in countries where alcohol consumption per capita exceeds the United States, which according to the World health Organization is 36 countries, the obesity rates do not follow the same pattern, which you’d expect if alcohol “pose[d] a major risk to those trying to keep their weight down.” According to WHO, Belarus, Andorra, Lithuania, Czech Republic, Grenada, Austria, Ireland, France, Saint Lucia, Estonia, Luxembourg, Germany, Russia, Slovakia, Portugal, Hungary, Croatia, Poland, Belgium, Denmark, Australia, the Bahamas, Slovenia, United Kingdom, Bulgaria, Switzerland, Spain, Latvia, Finland, New Zealand, the Netherlands, Gabon, Romania, Nigeria, Saint Kitts and Nevis and Cyprus all consume more alcohol per capita than the U.S., based on data for fifteen years, from 1990-2010.

And as for the most obese countries, we’re number one according to several sources, including Business Insider, the Telegraph and NationMaster. Although there are some sources that claim in 2013, Mexico took the title from us, yet it, too, is conspicuously absent from the list of countries that drink more than we do, meaning they drink less but are more obese.

Of those 36 countries that the WHO data makes clear drink more per capita than we do, only half of them appear on the OECD list of the top obese nations, from their 2012 Obesity Update report. If alcohol was causing people to eat more, than it seems clear people who drink more should likewise be eating more, too, and we’d see a direct correlation between both sets of numbers.

The three sources other than the WHO list also include on their lists of the most obese nations; Brazil, China, Colombia, Egypt, Iceland, India, Indonesia, Jamaica, Malaysia, Mauritania, Nauru, Nigeria, Pakistan, Qatar, Saudi Arabia, Spain, Sweden, Tonga, Turkey, UAE, and Zimbabwe, of which only two — Nigeria and Spain — drink more than we do. Again, if any of this were true, it seems obvious that there would be an easily recognizable correlation between both alcohol consumption and the obesity rates, but there isn’t, strongly suggesting there isn’t one at all.

I suspect the researchers know this, but the journalist who took the study and twisted it to fit a narrative probably did not. He finishes with this conclusion. “With America weighing down under an obesity epidemic and two out of every three American adults consuming alcohol, there is an immediate need to find more connecting factors between the brain, food, and alcohol, advise the scientists.” But is that what they’re advising? Because the evidence doesn’t quite measure up to that scary headline. If this were true, wouldn’t doctors be prescribing alcohol for their patients who need to eat more. I’d also say his article seems irresponsible, since it promotes an idea that it doesn’t actually support, and misrepresents the facts to get more people clicking on the link. It’s so bad that only a prohibitionist would fall for it, because facts don’t matter in propaganda, only making alcohol look bad.

Beer May Lessen Chronic Pain

Here’s another study you won’t see reported by Alcohol Justice, because it goes against their propagandist mantra. A study conducted at the University of Aberdeen in Scotland essentially found that the moderate consumption of alcohol might lessen chronic pain, especially in people with fibromyalgia, defined as a “a syndrome characterized by fatigue and chronic pain in the muscles and in tissues surrounding the joints.”

Drinks Business summarized the findings:

In a study of over 2,000 sufferers of chronic widespread pain, those who often consumed above average amounts of alcohol had lower levels of disability than those who never or rarely drank.

The research into sufferers of fibromyalgia — a rheumatic condition that causes muscular pain and stiffness — surveyed patient’s eating and drinking habits to determine the effect of diet on their symptoms.

Of the 2,239 people surveyed, those who drank 21 to 35 units of alcohol per week were 67% less likely than to experience disability than those who didn’t drink.

The study itself was published on the July issue of the journal Arthritis Care & Research under the title “Moderate alcohol consumption is associated with lower risk (and severity) of chronic widespread pain: Results from a UK population-based study.”

Aberdeen also put out a pdf with the basics of the study and here’s the Abstract:

Objectives: To determine whether reported level of alcohol consumption is associated with the likelihood of reporting chronic widespread pain (CWP) and, amongst persons with CWP, the associated disability.

Methods: A population-based study in two areas of the United Kingdom. Participants self-completed a postal questionnaire. They were classified according to whether they met the American College of Rheumatology definition of CWP and whether the pain was disabling (Chronic Pain Grade III or IV). They reported their usual level of alcohol consumption. Potential confounding factors on which information was available included age, gender, cigarette smoking, employment status, self-reported weight and height and level of deprivation.

Results: 13,574 persons participated (mean age 55 years; 57% female) of whom 2239 (16.5%) had CWP: 28% reported never regularly consuming alcohol, 28% consuming up to 5 units/wk, 20% 6-10 units/wk and 24% more than 10 units/wk. Amongst persons with CWP, disability was strongly linked to level of alcohol consumption. Prevalence of disability decreased with increasing alcohol consumption up to 35 unit/wk (Odds Ratio (OR)21-35 units alcohol/wk v. never drinkers 0.33 95% CI (0.19,0.58)) adjusted for confounders. A similar relationship was found between reporting CWP and level of alcohol consumption (adjOR21-35 units alcohol/wk v. regular drinkers 0.76 95% CI (0.61-0.94).

Conclusions: This study has demonstrated strong associations between level of alcohol consumption and CWP. However the available evidence does not allow us to conclude that the association is causal. The strength of the associations means that specific studies to examine this potential relationship are warranted.

So while the researchers believe more study is necessary to confirm a causal connection, they do believe there are “strong associations” between moderate drinking and chronic widespread pain, and that those are robust enough to warrant additional study.


Desperation Propaganda

The ink was barely dry on my last post about Alcohol Justice’s tenuous grasp on honesty where they claimed Craft Brewers Just Don’t Care when they did it again, with this tweet:


The funniest part of their tweet is the claim that the Carlsberg Group, the fourth largest beer company in the world, producing 6.2% of the world’s beer, is “desperate for market share” and therefore gave up on beer and decided to diversify into beauty products. This they apparently concluded from an article on Mashable entitled Men, stop drinking beer and start rubbing it on your face. As the article itself makes abundantly clear — but is virtually ignored by AJ — using beer or beer ingredients in health and beauty products has been around forever and is nothing new. There are almost too many instances to mention. Shampoo with beer in it has been around for years, if not centuries. Dogfish Head and many others have been making soap with beer for just as long. There’s nothing in the article about why they’re diversifying (though anyone with a working knowledge of how a business operates will say “well, duh,” diversification is almost always a good move). These came out of the Carlsberg Lab, which has been doing research on beer for over 100 years, and in fact the lager yeast known as Saccharomyces pastorianus, was also once known as Saccharomyces carlsbergensis because of work done by the brewery on yeast in the late 1800s and early 20th century. Here’s the The press materials for the new products.

So there’s absolutely nothing to suggest that Carlsberg’s Beer Beauty products have anything whatsoever to do with desperation. Alcohol Justice just made that up. Why? Because they have to turn a lighthearted story into something they can use as propaganda. Because the truth is not something they seem remotely interested in. If anything, I think it shows their own desperation in trying to find something to complain about.


Craft Brewers Just Don’t Care

Nobody can piss me off faster than Alcohol Justice, the self-styled do gooders and self-proclaimed “watchdogs” of those of us in the evil alcohol empire. I just noticed this yesterday, but for at least the last week, they’ve been tweeting this inaccurate and misleading message that “High-calorie craft beer maker’s don’t care.” Here’s what they say:


You don’t have to be Noam Chomsky to parse this sentence and figure out what they’re saying. Beer has lots of calories and brewers just don’t give a shit, painting craft brewers in a negative light, something Alcohol Justice has turned in to an art form. But is that the truth? Is that even what the source of this claims? Regular readers won’t be surprised to learn that AJ once again has taken a statement and twisted into something else to promote their own agenda. Shocking, I know. The original source of this comes from an article in the Columbus Dispatch on June 26 entitled Small breweries sometimes make high-octane, high-calorie beers.

It’s all about calories and how some higher alcohol beers made by craft brewers have more calories than lower alcohol beers, which is a world class “duh.” If you claim you don’t know that more alcohol has more calories then I’m amazed at the level of ignorance you’re willing to admit. If you’re drinking an imperial stout and have convinced yourself it contains no more calories than a pale ale, you’re willfully deluding yourself, and you probably know it, even if you won’t admit it. But I’m not terribly interested in the calorie question, it’s been done to death and certainly isn’t going away anytime soon. What really annoys me is Alcohol Justice’s flippant hatred of brewers and insisting they don’t care, as if that makes them bad people. The reality, of course, is quite different. Here’s the relevant portion of the article:

Unless they’re aiming for a low-cal beer to appeal to dieters, day drinkers and the like, craft brewers say they don’t give two pints about calories. They’re after flavor and aroma and other qualities that make drinking good beer worth it. The qualities of your favorite porter or citrus-forward IPA depend upon a series of ingredient choices and the complex interplay of water, grain, hops and yeast that follows.

So it’s not that the brewers “don’t care” about higher calories, it’s simply that they place more emphasis on aromas and flavors, preferring to create beers that feature those more prominently. They’re not willfully making high calorie beers just to fatten people up and make them unhealthy, as AJ suggests. And why pick on brewers? This is especially galling since wine and sprits, with far more alcohol, has … wait for it … far more calories. Beer has the fewest calories of any alcoholic drink by ounce. I’m sure people will argue that people drink more beer so that’s moot. But the point of drinking better beer is to drink less of it. To me at least, beer with flavor is not made to pound, but to enjoy at a more reasonable pace, usually determined by the circumstances. Imperial stout is not made to be swigged, but shared in a snifter or similar glass, so the idea that it’s the same as any other beer seems at best facetious. If you’re downing pint glasses of Parabola or Ten FIDY you’re doing it wrong. Here’s an infographic that accompanied the article.


But the larger point is why do we attack beer and alcohol makers for the natural amount of calories created by the way they’re made? We don’t do that for calorie-rich desserts like cake, ice cream and pie. You know what else is high in calories? Cereal, avocados, bananas, nuts and berries, granola bars, pasta, lobster and so many more foods we love. But we’re not lambasting the people who farm, grow or make those for not caring about how high in calories they are. That’s because they’re not intentionally making them high in calories, it’s just the result of their nature. The same is true for beer. Brewers aren’t intentionally making high-calorie beers to fuck with people the way Alcohol Justice so churlishly insists. They’re making them because they taste good, and people want them, not because they just “don’t care.”

Are there no ethical standards for non-profits? Shouldn’t “watchdogs” who claim the moral high ground have to at least be honest and truthful themselves? Because even though they claim “beer makers don’t care,” they certainly don’t seem to care about their own veracity, and instead twist anything they can to fit their increasingly narrow narrative that everything having to do with alcohol, and especially beer it seems, is bad.

The Most Distinctive Causes Of Death By State

This is somewhat interesting, though it was little to do with beer. The CDC released the results of an analysis of the “most distinctive cause of death for each state and the District of Columbia, 2001–2010.” I never realized this, but it makes sense. The CDC uses a standardized List of 113 Selected Causes of Death, based on the International Classification of Diseases, 10th Revision. This is to help the data collected be more useful, and allows comparisons to be drawn if the data is not affected by local bias or custom. Then the data used was “age-adjusted state-specific death rate for each cause of death relative to the national age-adjusted death rate for each cause of death, equivalent to a location quotient.”

The analysis that went into creating the map was done by Francis Boscoe, who’s a researcher at the New York State Cancer Registry. Here’s the main findings, from the CDC website:

The resulting map depicts a variety of distinctive causes of death based on a wide range of number of deaths, from 15,000 deaths from HIV in Florida to 679 deaths from tuberculosis in Texas to 22 deaths from syphilis in Louisiana. The largest number of deaths mapped were the 37,292 deaths in Michigan from “atherosclerotic cardiovascular disease, so described”; the fewest, the 11 deaths in Montana from “acute and rapidly progressive nephritic and nephrotic syndrome.” The state-specific percentage of total deaths mapped ranged from 1.8% (Delaware; atherosclerotic cardiovascular disease, so described) to 0.0005% (Illinois, other disorders of kidney).

Some of the findings make intuitive sense (influenza in some northern states, pneumoconioses in coal-mining states, air and water accidents in Alaska and Idaho), while the explanations for others are less immediately apparent (septicemia in New Jersey, deaths by legal intervention in 3 Western states). The highly variable use of codes beginning with “other” between states is also apparent. For example, Oklahoma accounted for 24% of the deaths attributable to “other acute ischemic heart diseases” in the country despite having only slightly more than 1% of the population, resulting in a standardized mortality rate ratio of 19.4 for this cause of death, the highest on the map. The highest standardized mortality rate ratio after Oklahoma was 12.4 for pneumoconioses in West Virginia.

A limitation of this map is that it depicts only 1 distinctive cause of death for each state. All of these were significantly higher than the national rate, but there were many others also significantly higher than the national rate that were not mapped. The map is also predisposed to showing rare causes of death — for 22 of the states, the total number of deaths mapped was under 100. Using broader cause-of-death categories or requiring a higher threshold for the number of deaths would result in a different map. These limitations are characteristic of maps generally and are why these maps are best regarded as snapshots and not comprehensive statistical summaries.

Notice that despite prohibitionists claiming that alcohol is the “3rd-Leading Preventable Cause Of Death,” it’s actually not even on the list. It’s not even on the list of 113, apart from the more specific “Alcoholic Liver Disease.” Also, cancer isn’t among any of the top cause for any individual state, which is surprising given that it’s usually listed as the number two cause overall. Some of the stranger ones include Oregon and Nevada, whose leading cause is “legal intervention.” Then there’s Alabama and Tennessee with “accidental discharge of firearms,” while in Arizona and Arkansas it’s “discharge of firearms, undetermined intent.” Is anyone else bothered by the fact that in four states you’re most likely to die by being shot, whatever the reason?

Where Do The Moderate Drinking Guidelines Come From?

For as long as I can remember, the recommended daily allowance to remain within moderate drinking guidelines has been one drink for a woman and two for a man. With the USDA’s new 2015 Dietary Guidelines open for comment, Modern Drunkard magazine, through their Brutal Hammer news blog, attempted to discover where those longstanding “2 for a man/1 for a woman” (2m/1w) guidelines came from, and wrote up their efforts in The CDC Is Stonewalling Us. In some ways it’s a silly piece, hinging on the CDC’s website comment apparatus not working, but the overriding question is sound. While the rest of the document about the Dietary Guidelines is heavily footnoted, with numerous references to the basis for their recommendations, the 2m/1w guidelines is suspiciously and conspicuously absent of any underlying scientific support.

Nowhere is it apparent how they came to that determination. No footnotes, no citations of scientific studies, not a damn bit of evidence to support it. Granted, my bourbon binoculars (the classier version of beer goggles, but they see deep into the truth of things) can only take in so much information at a time, but I couldn’t find a shred of reasoning for these arbitrary numbers.

I’d never thought about that before, but it’s a valid question. Where did they come up with that? And it’s not an unimportant one. The guidelines for defining moderate consumption are not the same worldwide, and in fact vary widely.

For example, Professor David J. Hanson at the State University of New York notes. “The fact that alcohol consumption guidelines are arbitrary is demonstrated by the wide variance in maximum limits recommended around the world. For example Poland’s recommended limit is 12.5 units per week whereas Australia’s is 35. Indeed, much research finds better health and greater longevity associated with drinking above the recommended guidelines published by most countries.” To contrast the U.S. guidelines, “Canada recommends that men on average consume no more than three drinks per day, five days per week, for a total of 15 drinks per week. For women it recommends, on average, no more than two drinks per day, five days per week, for a total of 10 drinks per week.”


A British examination of 27 European nation’s guidelines found “a remarkable lack of agreement about what constitutes harmful or excessive alcohol consumption on a daily basis, a weekly basis and when driving, with no consensus about the ratios of consumption guidelines for men and women.” Hanson concluded. “Thus, it appears that the differences in recommended guidelines are not based solely on the scientific medical evidence, but on cultural and political considerations. That is, the guidelines are highly arbitrary.”

And in some cases, capricious, as well. It was revealed in 2007, twenty years after the guidelines for the UK had been set in stone in 1987, that they were simply made up. One committee member who’d worked on the guidelines remembered that they were simply “plucked out of the air” and had “no basis in science” whatsoever, which I detailed at the time in Target: Alcohol. Without a clear basis on which our own guidelines were arrived upon, how can we be certain ours are any less fabricated inventions?

The other issue that’s never adequately addressed is the split for men and women. Supposedly, it’s because “Women tend to be smaller, but also have different body compositions and different metabolic enzymes.” But we know that weight matters. It’s how we figure out how much an individual can drink before they’ll be drunk or at least reach a specific blood alcohol level, because the rates are fairly precise when accounting for weight plus intake. So why do we ignore that simple knowledge with the guidelines? There are, of course, plenty of small, light men as well as many heavier women. It’s just a reality that people are diverse.

The International Center for Alcohol Policies or ICAP, somewhat disingenuously claims that the “Recommendations are based on scientific data regarding drinking levels at which risk increases,” yet never reveals where this “scientific data” comes from. And the fact that the guidelines vary widely from country to country would seem to suggest otherwise. Because if there was hard scientific data it would be the same everywhere, and the guidelines would not vary by as much as they do.

The closest thing I can find in the U.S. is at the Recommended Alcohol Questions on the NIH and NIAAA website states that the guidelines are “based on recent epidemiological studies on alcohol intake and risks which have demonstrated that for estimating risk of mortality, morbidity (including injuries) and other problems including drunk driving and social harms.” But then where are the citations for these epidemiological studies, and how could they possibly quantify such subjective issues as “social harms.” Quick answer: they can’t, not and remain purely scientific as the guidelines really should be.

I had never stopped to question the 2m/1w guidelines before, and it appears neither did almost anyone else. While there are plenty of citations for many aspects of the dietary guidelines, when it comes to alcohol, the government suddenly goes silent. But it doesn’t seem like too much to ask that the scientific basis for them be revealed and transparent. I’m not even arguing against them, and have always thought they were somewhat reasonable, especially in their current incarnation with the addition of the weekly limits. But we really should be able to see how they were arrived at, and what science, if any, they were based on.


USDA Dietary Guidelines Under Fire Again From Prohibitionists

Every five years, the U.S. Department of Agriculture, or USDA, in conjunction with the Department of Health and Human Services, or HHS, updates its quinquennial Dietary Guidelines. They’re described as providing “authoritative advice for Americans ages 2 and older about consuming fewer calories, making informed food choices, and being physically active to attain and maintain a healthy weight, reduce risk of chronic disease, and promote overall health.” Since the last guidelines were published in 2010, it’s time for the new ones, and they’ve been proposed and are are now open for comments before being finalized.

In the 2010 Guidelines, a change was made to the structure of the recommended amounts of alcohol people should consume, if they’re going to enjoy drinking alcohol and are, of course, of legal age. At the time, the government took the radical view, to prohibitionists, that:

The consumption of alcohol can have beneficial or harmful effects, depending on the amount consumed, age, and other characteristics of the person consuming the alcohol. Alcohol consumption may have beneficial effects when consumed in moderation. Strong evidence from observational studies has shown that moderate alcohol consumption is associated with a lower risk of cardiovascular disease. Moderate alcohol consumption also is associated with reduced risk of all-cause mortality among middle-aged and older adults and may help to keep cognitive function intact with age. However, it is not recommended that anyone begin drinking or drink more frequently on the basis of potential health benefits because moderate alcohol intake also is associated with increased risk of breast cancer, violence, drowning, and injuries from falls and motor vehicle crashes.

I may not agree with some of the characterizations in the last sentence, but it does serve to demonstrate how conservative the guidelines are, and that they’re not cavalierly telling people to start drinking. Plus, unlike some anti-alcohol groups, I’m not trying to willfully mislead people about what they say. They also have a handy chart of key definitions.


So what that second definition means is that if you’re a woman, you can enjoy 3 alcoholic drinks a day (or less), so long as you don’t have more than 7 during the same week, and you’ll be considered to not be a heavy drinker or engaging in high-risk drinking. A man, however, may enjoy 4 alcoholic drinks a day (or less), so long as he doesn’t have more than 14 during the same week, and he’ll likewise be considered to not be a heavy drinker or engaging in high-risk drinking. That, in effect, relaxed the “1 drink per day for women and up to 2 drinks per day for men” axiom that had been in place for a long while. When those changes made the rounds five years ago, the prohibitionists threw a temper tantrum and accused the government of all manner of bias and corruption, which is almost funny given how conservative they really are.

The new Dietary Guidelines for Americans, for 2015, are now going through the comment period, and once again the prohibitionists are apoplectic. Alcohol Justice, for example, whines that the government “proposes a risky and harmful shift in its definition of moderate drinking, and promotes drinking as a healthy dietary behavior. It suggests that a two-to-threefold increase in daily consumption limits is safe, and that questionable claims of health benefits outweigh known, substantiated risks of alcohol consumption. The Report represents a significant departure from previous Dietary Guidelines, and does so without sufficient scientific basis to justify such a shift.”

So how honest is that statement? Let’s take a look. First, what is the “risky and harmful shift in its definition of moderate drinking” from 2010 to 2015? The “new” language is on Page 105 of 107, constituting the proposed guidelines for 2015.

2015 Language:

Moderate alcohol consumption — Average daily consumption of up to one drink per day for women and up to two drinks per day for men, with no more than three drinks in any single day for women and no more than four drinks in any single day for men.

And here’s the old language below. Notice the difference? No? That’s because there really isn’t any. There are a few of the words that are different, numbers replaced by the word written out, some different punctuation, but that’s about it. The meaning is entirely the same.

2010 Language:

Moderate alcohol consumption is defined as up to 1 drink per day for women and up to 2 drinks per day for men. Heavy or high-risk drinking is the consumption of more than 3 drinks on any day or more than 7 per week for women and more than 4 drinks on any day or more than 14 per week for men.

There is no shift. If anything, this version of the guidelines merely confirms changes made to the 2010 Guidelines. “Regarding alcohol, the Committee confirmed several conclusions of the 2010 DGAC, including that moderate alcohol intake can be a component of a healthy dietary pattern, and that if alcohol is consumed, it should be consumed in moderation and only by adults. However, it is not recommended that anyone begin drinking or drink more frequently on the basis of potential health benefits.”

AJ says the “Dietary Guidelines should recommend ways to reduce and prevent alcohol-related harm, not increase it,” but of course that’s not at all what they say. That’s just more whining because they don’t like what the USDA is proposing. They didn’t like it five years ago, and they don’t like it now. They go on to claim that with “current and growing evidence regarding risk of disease and harm from drinking even low levels of alcohol, the Dietary Guidelines should include recommendations for Americans to drink less alcohol – not more.” Of course, that’s another misleading statement. They can, and often do, cite single studies that say what they want, but as detailed in Studies Show Studies Don’t Show Much, most are not worth the paper they’re printed on, but they keep hammering on them because it makes for effective propaganda, especially in the school of “if you repeat a lie often enough ….”

AJ further believes that the proposed guidelines say “that a two-to-threefold increase in daily consumption limits is safe.” But this mythical increase is just that, a fantasy. The 2010 guidelines said the same thing. There’s no proposed increase, just a confirmation of the last version. And guess what happened with the 2010 change? Nothing, that’s what. The country did not fall to ruin from people suddenly drinking too much because they believe the guidelines told people they should, or could.

Then they accuse the guidelines are based on “questionable claims of health benefits [which] outweigh known, substantiated risks of alcohol consumption. The Report represents a significant departure from previous Dietary Guidelines, and does so without sufficient scientific basis to justify such a shift.” What utter bullshit. Do you know what constitutes a “questionable claims of health benefits?” Anything that AJ doesn’t agree with. And how they define “known, substantiated risks of alcohol consumption?” That’s easy, it’s one they like that agrees with their skewed world view. As shown, this is absolutely NOT “a significant departure from previous Dietary Guidelines,” but is virtually identical to the 2010 version. And their statement that there is not “sufficient scientific basis to justify such a shift” is laughable because they’ll never except any scientific evidence that disagrees with or contradicts their dogma. Here’s how the USDA explains how they arrived at the alcohol guidelines.

As alcohol is a unique aspect of the diet, the DGAC considered evidence from several sources to inform recommendations. As noted above, moderate alcohol intake among adults was identified as a component of a healthy dietary pattern associated with some health outcomes, which reaffirms conclusions related to moderate alcohol consumption by the 2010 DGAC.

No matter how you slice it, there is nothing new regarding the alcohol guidelines in the proposed dietary guidelines for 2015. But to hear Alcohol Justice tell it, this is “a radical change,” despite being almost exactly the same as five years ago. This is their action plan for the faithful sheep that follow them, [with my rebuttal in brackets]:


Without providing any explanation or evidence for a radical change [they do explain the reasons, citing that there’s evidence supporting their decision], the Dietary Guidelines Advisory Committee proposes to increase limits used to define “moderate” drinking. [No, they don’t. All they do is confirm the changes made five years ago.]

The current (2010) U.S. Dietary Guidelines define moderate drinking as up to 1 drink per day for women and up to 2 drinks per day for men (daily limits) [that’s not all they say, they also cite the weekly allowances]. In contrast, the proposed change would base these 1/2 limits on average rather than daily consumption and suggests it is safe for women to drink up to 3 drinks in a day and men up to 4 drinks in a day so long as the averages are not exceeded [exactly as they did in 2010]. This effectively triples (the daily limit for women and doubles (the daily limit for men). [Not this time, it doesn’t.]

Furthermore, the report implies that drinking is recommended as part of a healthy lifestyle: “the U.S. population should be encouraged to consume dietary patterns that are rich in vegetables …; moderate in low and non fat dairy and alcohol (adults).” [Oh, no! The horror. Frankly, what’s more surprising is that, given their findings that total mortality is improved with the moderate consumption of alcohol, they’re so conservative in their suggestions. But it makes sense in the context of anti-alcohol groups that throw tantrums any time their world view is challenged. But their statement that “the report implies that drinking is recommended as part of a healthy lifestyle” is complete and utter nonsense, and could even be called grandstanding because the language of the proposed 2015 guidelines also includes this: “However, it is not recommended that anyone begin drinking or drink more frequently on the basis of potential health benefits.” So it’s pretty crystal clear that the USDA is not recommending people start drinking “as part of a healthy lifestyle.” AJ just made that up.]


Since most adult drinkers in the U.S. don’t drink every day, the proposed change effectively encourages consumption right up to binge drinking levels, thus increasing health risk. [That identifies the problem with the definition of binge drinking, as I’ve written about numerous times. That’s the problem here, not encouraging people to drink moderately. After all, if they did, they might live longer. We wouldn’t want people to know that, would we?]

Binge drinking (4 or more drinks per occasion for women; 5 or more drinks per occasion for men) causes more than half of all alcohol related deaths each year in the U.S., and impairment and increased risk begin below those levels. The proposed changes are, therefore, dangerous for public health. [Again, that’s a problem with the definition of bingeing, which used to be more vague, making it hard to quantify. So it’s been narrowed over the years, and made easier to quantify, bringing more and more people into the specter of binge drinkers, artificially inflating statistics about its dangers.]

There are no randomized studies showing any health benefits from any level of alcohol consumption as well as no evidence that moderate drinking promotes a healthy lifestyle. [Poppycock. They’re hanging their hat here, one presumes, on “randomized” studies, but it’s unlikely even that’s true. The USDA itself in 2010, looked at meta-analysis of a wide range of studies, concluding just the opposite of AJ’s position. But AJ keeps ignoring that “evidence” because they don’t like it. It’s easier to just keep saying what they want to be true.]

It’s hard to know what to make of so dishonest a piece of propaganda as this is, raising unfounded fears, not to mention being littered with just out and out misinformation. It’s one thing to be in favor of promoting “evidence-based public health policies and organiz[ing] campaigns with diverse communities and youth against the alcohol industry’s harmful practices” but quite another to watch how that plays out in reality. “Evidence-based” seems to really mean anything they agree with and “the alcohol industry’s harmful practices” includes literally every single thing we do. I wish that was hyperbole, but I’ve never seen any action taken by an alcohol company that AJ didn’t find fault with, from donating cans of water to Haiti after the devastating earthquake there to their “‘charge-for-harm’ approach, which is based on the assumption that anyone who drinks deserves to be punished.” And another similar group stated at a 2013 conference that “they simply didn’t care about the public health impacts of taxes. They were in the game solely to get some of the tax revenue steered toward their organization.”

This is getting seriously out of hand. as anti-alcohol groups get bolder and more obviously prohibitionist, their divisiveness makes any meaningful discussion increasingly impossible. And unlike these prohibitionists, most people I know in the beer world, and the real world for that matter, recognize that while moderate drinking of alcohol is a good thing for a majority of adults, it’s not for everybody. Some people can’t handle it, and they often ruin it for the rest of us. Because those are the people that anti-alcohol folks always use to represent everyone who drinks, ignoring that they’re minority and that most of us can have a few drinks and not plunge the world into turmoil. But as long as they keep painting us as all the same, they’ll never be able to admit anything but an absolutist view of drinking, no matter how ridiculous that is, and no matter how ridiculous it makes them seem. When you start accusing the conservative USDA of ignoring science and encouraging people to start drinking, you’ve definitely jumped the shark.

Studies Show Studies Don’t Show Much

If there’s one thing prohibitionists love to shout about, it’s a new study showing how terrible alcohol is, and how it supports what they’ve been proselytizing about all along. A growing trend has been anti-alcohol groups funding studies, having the “team” look for problems through phrasing the study’s goals and methodology with a particular outcome in mind, and then releasing the results as if it was impartial news. Sadly, with our media overworked and underpaid, many fall for it and report such a sham study’s results without ever critically examining it or even looking for a dissenting opinion to bring some fair and balanced perspective. Prohibitionists, knowing this, package their press releases into print-friendly versions so media outlets can simply cut and paste, passing it off as actual news. To be fair, it’s not just them. Almost everybody does it. It’s become a game, of sorts, one where most reasonable people’s wishes are ignored in favor of a more extreme agenda. Issues get polarized, and meaningful dialogue is becoming increasingly impossible with mud being slung in both directions, though I tend to think on the prohibitionist front that more mud comes our way, than vice versa.

But I’ve spent the last decade or so taking a fairly critical look at study after study, taking issue with almost all of them in one way or another. For every study that says one thing, you can find another that says the complete opposite, which you’d think wouldn’t, or shouldn’t, be possible. But a lot of it has to do with the way studies are conducted, how rigorous the science is, and whether or not they started with a specific agenda or not. I’ve certainly crowed about studies that show alcohol in a positive light, though I’ve never financed any. But despite all the tamper tantrums from the prohibitionists, they’re the ones spending all the money creating a false record of harm, not to mention taking advantage of any others they can, part of their post-prohibition strategy to bring down alcohol by less obvious means in a slower, more patient approach, chipping away at public policy and the law brick by brick, so to speak.

As a result of seeing so many of these so-called “studies,” I’ve noticed a lot of tricks that they use to make them seem like the findings actually mean something, but they rarely do, and usually even the study’s authors, who presumably want to keep their status as impartial scientists despite taking money for funding, almost always issue cautions and calls for further testing and for no one to make too much of what they found, words invariably ignored by the people using their findings to promote an agenda. It’s made me question the entire medical, and to some extent the scientific, community because it’s so obviously been corrupted by money — like every other aspect of our society, sad to say — with so many willing to take money to help a fanatical group promote its agenda. And it seems like the shear number of such studies has ballooned in recent years, too. Just how many scientific journals can there be, and how many are truly scientific, if any?

But an article on Vox a few days ago addressed this very issue, with This is why you shouldn’t believe that exciting new medical study. As the author wonders “whether there is any value in reporting very early research,” I’ve seen how it’s more often misused than anything else. As she writes. “Journals now publish their findings, and the public seizes on them, but this wasn’t always the case: journals were meant for peer-to-peer discussion, not mass consumption.” Because of this, the amount of studies being conducted has skyrocketed since their use is often now well beyond the original purpose of real study and furthering the science surrounding an issue. The actual number of so-called journal studies have seen an astounding 300% increase over the last quarter-century.


But as she points out, early reporting on these studies rarely leads to any meaningful breakthroughs, even though those initial findings become fixed in the public mind as fact. A recent example that springs to mind is about glutens. A study in Australia initially seemed to suggest that eating gluten-free could be healthier for even people who didn’t suffer from Celiac disease, but further work by the same scientist found that his initial results were incorrect, and that there were no appreciable health benefits to a gluten-free diet for most people. Despite this clear repudiation of the initial findings, gluten-free as a healthier lifestyle remains an idea many people not only still believe, but even follow, despite having been refuted years ago. This is not an isolated occurrence.

In 2003, researchers writing in the American Journal of Medicine discovered something that should change how you think about medical news. They looked at 101 studies published in top scientific journals between 1979 and 1983 that claimed a new therapy or medical technology was very promising. Only five, they found out, made it to market within a decade. Only one (ACE inhibitors, a pharmaceutical drug) was still extensively used at the time of their publication.


So that means 100 others proved to not pan out, their promise as originally reported proving to not stand up to further research or lead to any meaningful breakthrough. But the news cycle has already moved on, and the damage has been done, with the study reported and its inaccurate findings fixed into people’s minds. And this is just one of the reasons why immediately promoting the results of a study to the public is a bad idea. As the Vox article makes clear. “This cycle recurs again and again. An initial study promises a miracle. News stories hype the miracle. Researchers eventually disprove the miracle.”

“There’s a big, big, difference between how the media think about news and how scientists think about news,” Naomi Oreskes, a Harvard professor of the history of science, recently told [Vox’s Julia Bellus] in an interview. “For you, what makes it news is that it’s new — and that creates a bias in the media to look for brand new results. My view would be that brand new results would be the most likely to be wrong.”

In some cases, results are published too soon precisely to get attention for the study or the research in order to get more funding to carry on the research, or simply because of the pressure to “publish or perish” in academia or a career. Or, of course, it’s published specifically to promote an agenda or ideology.


More often than not, single studies contradict one another — such as the research on foods that cause or prevent cancer. The truth can be found somewhere in the totality of the research, but we report on every study in isolation underneath flip-flopping headlines. (Red wine will add years to your life one week, and kill you quicker the next.)

This is seen in beer, a lot, too. But as the graph below makes clear, it happens everywhere, all the time, with the main culprit being the media in general, and the prohibitionists more specifically, reporting on single studies that show one thing rather then treating the issue as a whole or continuum of understanding. In particular, Alcohol Justice frequently takes one study that shows something in line with their agenda and treats it as if it’s the final answer and no further study is necessary; they’re right, case closed. Which, as you can see, is never the case.


A good example of this is a recent tweet from Alcohol Justice, questioning that “Alcohol good for your heart? Evidence is evaporating Don’t believe industry-sponsored science.”


The link takes you to a USA Today story, entitled Alcohol good for your heart? Evidence is evaporating, which is where AJ got the witty language in the tweet. But the part about not believing “industry-sponsored science” is completely made up. The story never even addresses that as an issue. It’s pure propaganda. As you’ll see, the trail from the USA Today story leads not to “industry-sponsored science,” but to another anti-alcohol group.

The USA Today story itself is a hodgepodge of misinformation and innuendo, written in that most common style of the mainstream media that believes scaring people captures their attention and gets ratings, viewers or whatever metric they measure their success by. Early in the piece, the author sets out her premise.

But before you pour your next cocktail, beer or glass of wine, you should know this: the science suggesting a benefit has never been conclusive. And some experts believe the evidence is getting thinner all the time.

Almost no science is conclusive, or ever has been. That’s the point of continuously conducting research, to constantly learn more and to further our understanding of whatever’s being studied. But just as benefits may be inconclusive, the evils are similarly inconclusive. But she chose to frame the story in such a way as to emphasize the negative, despite the fact that the statement could be said almost any way and still be technically correct. And saying “some experts” reveals that not everyone agrees, even with so vague a premise. You can always find a person to disagree about anything, especially if they have some reason to do so.

To illustrate what I mean, take her reliance on an editorial written by “Mike Daube, professor of health policy at Curtin University in Australia.” He “writes that the once-touted benefits of moderate drinking ‘are now evaporating,'” providing the piece’s catch phrase and hook. But who is Mike Daube. Is he a doctor or scientist? Nope. Is he an impartial expert? Hardly, “Mike Daube, professor of health policy” is all that USA Today reports, and at the editorial she’s quoting from, the only author affiliation listed is “Curtin University, GPO Box U1987, WA 6845, Australia.”

But you don’t have to look too hard to find out that Mike is also co-chair of Australia’s National Alliance for Action on Alcohol, an organization who’s sole stated purpose is that is “has been formed with the goal of reducing alcohol-related harm.” So while he’s railing against “industry lobbying and promotion [being] rife and unchecked by governments, he’s pretending to be an impartial health professional, while also leading an organization who’s already convinced that alcohol has only a negative impact on society and is working to battle it, or get rid of it. That doesn’t seem particularly impartial to me. How utterly disingenuous and hypocritical. He’s using his background as a health policy professor to make it seem as if he has some expertise in medicine, but his area of study is public policy, with an emphasis on health, and you don’t need an advanced degree to understand those are two very different things.

And the editorial USA Today is relying on, Alcohol’s evaporating health benefits (they sure love a good turn of phrase, don’t they?), is published in The BMJ (formerly the British Medical Journal). So essentially a policy expert — who in 2012 was “awarded [the] ‘Oscar’ of public health campaigning — is editorializing about science and medicine in a medical journal. It’s an editorial — an opinion. No matter how authoritative, he should carry about as much weight on scientific matters as I do. We have exactly the same number of doctoral degrees in medicine.

Even so, while lambasting alcohol over a new British study which forms the basis for his “evidence is evaporating” quip, he has to admit that the study did show a positive correlation for “women aged 65 or more” but dismisses that as “at best modest and likely to be explained by selection bias.” Which may true, but then again maybe not. Perhaps more study is necessary before making such sweeping pronouncements as the “evidence is evaporating.” Which is entirely the point. He’s looking at one study in a vacuum and choosing the outcome he favors, because of his own bias. So that’s not, or should not, be newsworthy. “Hey guess what? What I believed all along is what I still believe, and here’s this one study that partially agrees with me, so I must be right after all. Can I be in your scientific journal?” Is this really what passes for peer-reviewed science? What a load of bollocks.

The USA Today article is actually very short, but is padded out with a list of “what U.S. experts say you need to know for now.” Unfortunately, that list is entirely about the negative aspects of alcohol consumption and completely ignores any positive contributions to a person’s health, and it’s not like they’re hard to find.

But one study said something different, so I guess all those others are wrong, right? Yet this is the approach prohibitionist groups take time and time again. And as the Vox article makes clear, this approach can result in creating false hopes and leading researchers, scientists and even public policy-makers down the wrong path. As journalist Julia Belluz admits, it’s hard for the press to not jump at new study results, because their novelty is catnip to the management structure of both old and new media. But as the media blinders are understandable and even forgivable, at least to some extent, that’s not the case for the anti-alcohol groups who take that news and use it unscrupulously to advance their agenda. They’re the ones doing actual harm, because they’re creating a false narrative that is dishonest and knowingly wrong. I think they’ve forgotten that advancing a particular point of view doesn’t mean destroying the other side by any means possible, especially since they so often claim to own the moral high ground. But if their “ends justify the means” strategy reveals anything, it’s that they don’t own a mirror. They only judge our morals, attacking us frequently and accusing us of caring only about business, money and hurting children.

The Vox article concludes with some sage advice from “Harvard’s Oreskes, Stanford’s John Ioannidis, and many other respected researchers,” who insist “we need to look past the newest science to where knowledge has accumulated. There, we’ll find insights that will help us have healthier lives and societies.” Could somebody please tell the prohibitionists?

New Study Shows Chemical In Beer Prevents Alzheimer’s And Parkinson’s

A new study conducted in China suggests that “beer is good for the brain.” According to to an article in the Inquisitr, here’s why. “The beer draws its superpowers from hops, the female flowers of the hop plant Humulus lupulus, which are used primarily as a flavoring and stability agent in beer. However, apart from contributing to the signature taste of the beer, hops releases a chemical — Xanthohumol — that has the potential to fight off neurodegenerative disorders such as Alzheimer’s and Parkinson’s disease.” These findings come from a journal article, with the decidedly unsexy title Xanthohumol, a Polyphenol Chalcone Present in Hops, Activating Nrf2 Enzymes To Confer Protection against Oxidative Damage in PC12 Cells, which was published in the Journal of Agricultural and Food Chemistry, published by the American Chemical Society.


Here’s the abstract:

Xanthohumol (2′,4′,4-trihydroxy-6′-methoxy-3′-prenylchalcone, Xn), a polyphenol chalcone from hops (Humulus lupulus), has received increasing attention due to its multiple pharmacological activities. As an active component in beers, its presence has been suggested to be linked to the epidemiological observation of the beneficial effect of regular beer drinking. In this work, we synthesized Xn with a total yield of 5.0% in seven steps and studied its neuroprotective function against oxidative-stress-induced neuronal cell damage in the neuronlike rat pheochromocytoma cell line PC12. Xn displays moderate free-radical-scavenging capacity in vitro. More importantly, pretreatment of PC12 cells with Xn at submicromolar concentrations significantly upregulates a panel of phase II cytoprotective genes as well as the corresponding gene products, such as glutathione, heme oxygenase, NAD(P)H:quinone oxidoreductase, thioredoxin, and thioredoxin reductase. A mechanistic study indicates that the α,β-unsaturated ketone structure in Xn and activation of the transcription factor Nrf2 are key determinants for the cytoprotection of Xn. Targeting the Nrf2 by Xn discloses a previously unrecognized mechanism underlying the biological action of Xn. Our results demonstrate that Xn is a novel small-molecule activator of Nrf2 in neuronal cells and suggest that Xn might be a potential candidate for the prevention of neurodegenerative disorders.

This is not the first time such findings have been studied, so this appears to be yet another confirmation in the growing body of positive health benefits of moderate beer drinking. What the team of Chinese scientists found was a “previously unrecognized mechanism underlying the biological action of Xn,” suggesting Xanthohumol “might be a potential candidate for the prevention of neurodegenerative disorders.”

The Inquisitr concludes:

Hops have been used in Chinese medicine for centuries. However, its efficacy to prevent Alzheimer’s and Parkinson’s was discovered only recently. Neuronal cells — which are in the brain, spine, and nerves — are in limited supply over one’s lifetime. These cells are especially susceptible to stress. This stress is thought to be one of the ways brain-related disorders such as Alzheimer’s and Parkinson’s begin.

Beer, as I probably don’t need to remind you, is at least one great way to relieve stress.

Patent No. 2452476A1: Mediating The Effects Of Alcohol Consumption By Orally Administering Active Dry Yeast

Today in 2003, US Patent 3712820 A was issued, an invention of Joe Owades, for their “Mediating the Effects of Alcohol Consumption by Orally Administering Active Dry Yeast.” Here’s the short Abstract. “A process for lowering blood alcohol levels in humans after they imbibe alcoholic beverages by administering active dry yeast before or concomitantly with the imbibing of the beverages.”

This is most likely the origin of the hangover prevention that Jim Koch, from the Boston Beer Co., has popularized over the years, but especially after Esquire magazine ran an article about it last April, How to Drink All Night Without Getting Drunk.


The story got picked up by NPR, Serious Eats and even Snopes took a look at it.

But I’d actually heard Jim tell the story a couple of times at various events, most recently at a beer dinner last year at the Jamaica Plain brewery in Boston celebrating the 30th anniversary of Samuel Adams.


In telling the story, Jim did, of course, mention that the idea came from Joe Owades, who had worked as a consultant with the Boston Beer Co. since the very beginning, and off and on thereafter. But I don’t think I’d realized before now that Joe had actually patented the idea.

The claim in the patent application describes it in a nutshell. “A method of mediating the effect of alcohol consumption by a person which comprises orally administering active dry yeast containing alcohol dehydrogenase to said person prior to or simultaneously with consumption of an alcohol-containing beverage, whereby to oxidize a portion of the alcohol while still in the stomach of said person.” His own testing of the method, shown in the figures below, found that “blood alcohol level-min. was reduced by 38% by the yeast.”